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Upregulation of Soluble HLA-G in Chronic Left Ventricular Systolic Dysfunction
Line Lisbeth Olesen
,
Thomas Vauvert F. Hviid
*
*
Corresponding author for this work
Cardiology
Clinical Biochemistry
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Explore the research areas of 'Upregulation of Soluble HLA-G in Chronic Left Ventricular Systolic Dysfunction'.
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Keyphrases
Ejection Fraction
100%
Soluble HLA-G
100%
Left Ventricular Systolic Dysfunction
100%
Uric Acid
50%
B-type Natriuretic Peptide
50%
N-terminal Fragment
50%
Blood Plasma
25%
Receiver Operating Characteristic Curve
25%
SNP
25%
Acid Value
25%
Human Leukocyte antigen-G (HLA-G)
25%
Neuroscience
Uric Acid
100%
Amino Terminal Sequence
100%
Brain Natriuretic Peptide
100%
Haplotype
50%
Blood Plasma
50%
Immunology and Microbiology
Upregulation
100%
Heart Ejection Fraction
100%
Amino Terminal Sequence
50%
Natriuretic Peptide
50%
Blood Plasma
25%
Single Nucleotide Polymorphism
25%
Haplotype
25%
Pharmacology, Toxicology and Pharmaceutical Science
Left Ventricular Systolic Dysfunction
100%
Brain Natriuretic Peptide
50%
Uric Acid
50%
Biological Marker
25%