Cardiorespiratory fitness, body composition, diabetes, and longevity: A 2-Sample Mendelian Randomization Study

Alisa D Kjaergaard*, Christina Ellervik, Niels Jessen, Sarah J Lessard

*Corresponding author af dette arbejde

Publikation: Bidrag til tidsskriftArtikelForskningpeer review

Abstract

CONTEXT: Cardiorespiratory fitness, commonly assessed as maximal volume of oxygen consumption (VO2max), has emerged as an important predictor of morbidity and mortality.

OBJECTIVE: We investigated the causality and directionality of the associations of VO2max with body composition, physical activity, diabetes, performance enhancers, and longevity.

METHODS: Using publicly available summary statistics from the largest genome-wide association studies publicly available, we conducted a bidirectional two-sample Mendelian randomization (MR) study. Bidirectional MR tested directionality, and estimated the total causal effects, whereas multivariable MR (MVMR) estimated independent causal effects. Cardiorespiratory fitness (VO2max) was estimated from a submaximal cycle ramp test (N≈90,000) and scaled to total body weight, and in additional analyses to fat-free mass (mL/min/kg).

RESULTS: Genetically predicted higher (per one standard deviation increase) body fat percentage was associated with lower VO2max (β=-0.36 [95% CI: -0.40, -0.32], p=6E-77). Meanwhile, genetically predicted higher appendicular lean mass (0.10 [0.08,0.13] p=3E-16), physical activity (0.29 [0.07,0.52]), and performance enhancers (fasting insulin, hematocrit, and free testosterone in men) were all positively associated with VO2max (p<0.01). Genetic predisposition to diabetes had no effect on VO2max. MVMR showed independent causal effects of body fat percentage, appendicular lean mass, physical activity, and hematocrit on VO2max, as well as of body fat percentage and type 2 diabetes (T2D) on longevity. Genetically predicted VO2max showed no associations.

CONCLUSION: Cardiorespiratory fitness can be improved by favorable body composition, physical activity, and performance enhancers. Despite being a strong predictor of mortality, VO2max is not causally associated with T2D or longevity.

OriginalsprogEngelsk
Artikelnummerdgae393
Sider (fra-til)1451-1459
Antal sider9
TidsskriftThe Journal of clinical endocrinology and metabolism
Vol/bind110
Udgave nummer5
Tidlig onlinedato12 jun. 2024
DOI
StatusUdgivet - maj 2025

Finansiering

BevillingsgivereBevillingsgivernummer
Novo Nordisk FoundationNNF22OC0076023
Boston Children's Hospital
National Institute of Diabetes and Digestive and Kidney Diseases R01 DK124258, R01 DK129850

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